OI not simply suppressed lipopolysaccharide-induced proinflammatory cascades of inducible nitric oxide synthase, cyclooxygenase-2, and cytokines launch in mouse BV2 microglial cells and also activated the Nrf2 signaling pathway and its downstream targets in these cells. Conditioned medium derived from OI-dealt with BV2 cells secured from rotenone- and MPP+-induced neurotoxicity in https://butylphthalide00986.theobloggers.com/35855248/about-sr-18292